Macrophages are the predominant cell type in chronic inflammation, contributing to tissue repair and the immune response.

Epithelioid cell formation is a characteristic feature of granulomatous inflammation, often seen in conditions like tuberculosis.

Neutrophils are the first responders to sites of acute inflammation, playing a crucial role in the initial immune response.

Fibrosis is the primary mechanism of tissue repair, involving the deposition of collagen and extracellular matrix components.

Histamine plays a key role in increasing vascular permeability and causing vasodilation during the inflammatory response.

Suppurative inflammation is characterized by the formation of pus, which consists of dead neutrophils, bacteria, and tissue debris.

IL-1 is a key cytokine that induces fever by acting on the hypothalamus during the inflammatory response.

Aspergillus species are commonly associated with chronic granulomatous disease due to the impaired ability of phagocytes to kill certain pathogens.

C-reactive protein (CRP) is a sensitive marker for inflammation and is commonly used in clinical practice.

Tissue necrosis is not a feature of acute inflammation; rather, it can be a consequence of prolonged inflammation or injury.